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Disease Info Card
Hypertrichosis
Information about Hypertrichosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypertrichosis
Most recent studies have shown that Hypertrichosis shares some biological mechanisms with acne, congenital-abnormality, dermatologic-disorders, diabetes-mellitus, disorders-of-porphyrin-metabolism, edema, gingival-hyperplasia, hair-diseases, hyperpigmentation, hyperplasia, hypertensive-disease, malignant-neoplasms, melanocytic-nevus, neoplasms, pigmentation-disorders, polycystic-ovary-syndrome, skin-neoplasms, virilism.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypertrichosis, and have been seen in publications frequently: Anagen, Anaphylaxis, Cell Proliferation, Coagulation, Excretion, Glomerular Filtration, Hair Cycle, Keratinization, Localization, Menarche, Menstruation, Muscle Atrophy, Ovulation, Pathogenesis, Pigmentation, Reflex, Regeneration, Secretion, Telogen, Wound Healing
Quite a number of genes have been found to play important roles in Hypertrichosis, such as BRD2, C2, CSH1, EGFR, ERCC8, FECH, HSPA9, INS, PLOD1, POMC, PRL, REN, SHBG, SLC29A3, UROD, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.