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Disease Info Card
Hepatic Ischaemia
Information about Hepatic Ischaemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hepatic Ischaemia
Most recent studies have shown that Hepatic Ischaemia shares some biological mechanisms with hemorrhage, hepatic-necrosis, hypoxia, inflammation, inflammatory-response, injury-to-liver, ischemia, liver-damage, liver-diseases, liver-failure, liver-failure-acute, liver-neoplasms, liver-regeneration-disorder, neoplasms, reperfusion-injury, stenosis, tissue-adhesions, warm-ischemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hepatic Ischaemia, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Cycle, Cell Death, Coagulation, Complement Activation, Cytokine Production, Excretion, Hepatocyte Proliferation, Inflammatory Response, Liver Regeneration, Neutrophil Activation, Oxidative Phosphorylation, Pathogenesis, Platelet Aggregation, Regeneration, Secretion, Transport, Transposition, Vasoconstriction
Quite a number of genes have been found to play important roles in Hepatic Ischaemia, such as CASP3, CAT, EDN1, HMOX1, IL10, IL6, ISYNA1, JUN, MPO, NFKB1, NOS2, NOS3, SLC17A5, SOD1, TLR4, TNF, XDH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.