Heparin-induced Thrombocytopenia (hit)

Overview of Heparin-induced Thrombocytopenia (hit)

Most recent studies have shown that Heparin-induced Thrombocytopenia (hit) shares some biological mechanisms with acute-coronary-syndrome, arterial-thrombosis, blood-coagulation-disorders, cerebrovascular-accident, deep-vein-thrombosis, embolism, gangrene, hemorrhage, heparin-induced-thrombocytopenia, infarction, kidney-failure, myocardial-infarction, pulmonary-embolism, purpura-thrombocytopenic-idiopathic, thromboembolism, thrombophilia, thrombosis, venous-thromboembolism, venous-thrombosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Heparin-induced Thrombocytopenia (hit), and have been seen in publications frequently: Aging, Anaphylaxis, Blood Coagulation, Cell Activation, Cell Adhesion, Coagulation, Endothelial Cell Activation, Fibrinolysis, Hemostasis, Hypersensitivity, Immune Response, Monocyte Activation, Pathogenesis, Platelet Activation, Platelet Aggregation, Proteolysis, Response To Heparin, Secretion, Sensitization, Sulfation

Quite a number of genes have been found to play important roles in Heparin-induced Thrombocytopenia (hit), such as ACOT7, ACTG1, ACTG2, APBB3, CPB1, F10, F2, F3, FHL5, MSR1, PF4, PPBP, SELP, SERPINA3, SERPINA5, SERPINC1, SRA1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Heparin-induced Thrombocytopenia (hit) Related Genes

click to see detail information for each gene

Pathways Related to Heparin-induced Thrombocytopenia (hit)

This information is being compiled and will come in a future update

Related Diseases