Disease Info Card

Hemorrhagic Colitis

Information about Hemorrhagic Colitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Hemorrhagic Colitis

Most recent studies have shown that Hemorrhagic Colitis shares some biological mechanisms with abdominal-pain, cattle-diseases, colitis, diarrhea, diarrhoea-haemorrhagic, escherichia-coli-infections, experimental-autoimmune-encephalomyelitis, gastrointestinal-hemorrhage, hemolytic-uremic-syndrome, hemorrhage, infective-disorder, kidney-failure, pain, purpura, purpura-thrombotic-thrombocytopenic, shigella-infections, thrombocytopenic-purpura, ulcer, ulcerative-colitis, uremia-syndrome.

Among the many pathways, these few ones have gauged particular interests from scientists studying Hemorrhagic Colitis, and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Division, Coagulation, Cytolysis, Dna Repair, Excretion, Fermentation, Glomerular Filtration, Immune Response, Inflammatory Response, Localization, Mucosal Immune Response, Pathogenesis, Platelet Aggregation, Quorum Sensing, Secretion, Swimming, Transport, Virulence

Quite a number of genes have been found to play important roles in Hemorrhagic Colitis, such as AKAP5, CFH, CRK, CRP, GUSB, HRH4, HSPB8, IL6, MAPK1, MAPK14, MCHR2, ST8SIA2, STX1A, STX2, TNF, UBN1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.