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Disease Info Card
Gout Acute
Information about Gout Acute: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Gout Acute
Most recent studies have shown that Gout Acute shares some biological mechanisms with arthritis, arthritis-gouty, arthropathy, chronic-gouty-arthritis, degenerative-polyarthritis, edema, gout, hypertensive-disease, hyperuricemia, inflammation, kidney-diseases, kidney-failure, maple-syrup-urine-disease, pain, primary-gout, rheumatism, rheumatoid-arthritis, sore-to-touch.
Among the many pathways, these few ones have gauged particular interests from scientists studying Gout Acute, and have been seen in publications frequently: Acute Inflammatory Response, Aging, Cell Activation, Cell Cycle, Cell Migration, Chemokine Production, Cytokine Production, Cytokine Secretion, Endothelial Cell Activation, Excretion, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Neutrophil Activation, Pathogenesis, Phagocytosis, Platelet Activation, Secretion, Transport
Quite a number of genes have been found to play important roles in Gout Acute, such as BCKDHA, CCL2, CRP, CYP3A4, IL1B, IL6, INS, JUN, MAPK3, NLRP3, POMC, PTGS2, TNF, TREM1, XDH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.