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Disease Info Card
Fraxe Syndrome
Information about Fraxe Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fraxe Syndrome
Most recent studies have shown that Fraxe Syndrome shares some biological mechanisms with ataxia, ataxia-spinocerebellar, atrophy, chromosome-fragility, developmental-delay-(disorder), dystrophy, fragile-x-syndrome, fraxa-syndrome, genetic-diseases-inborn, hereditary-diseases, huntington-disease, mild-mental-retardation, muscular-atrophy, myotonic-dystrophy, neurodegenerative-disorders, spinal-and-bulbar-muscular-atrophy-x-linked-1, trinucleotide-repeat-expansion.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fraxe Syndrome, and have been seen in publications frequently: Cell Cycle, Cell Death, Cell Growth, Cellular Process, Chromatin Remodeling, Chromosome Breakage, Dna Replication, Histone Methylation, Interphase, Localization, Methylation, Mitosis, Nucleosome Assembly, Oncogenesis, Organ Development, Pathogenesis, Regulation Of Translation, S Phase, Translation, Transposition
Quite a number of genes have been found to play important roles in Fraxe Syndrome, such as ABCG2, AFF1, AFF2, AFF3, AFF4, AR, ATN1, ATXN1, CBL, DLG3, DMPK, FDXR, FMR1, FRA10AC1, GAA, HTT, IDS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.