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- Table of Contents
, No Gclids
Disease Info Card
Foot-drop
Information about Foot-drop: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Foot-drop
Most recent studies have shown that Foot-drop shares some biological mechanisms with cerebrovascular-accident, flexed-fetal-attitude, foot-deformities-acquired, foot-diseases, fracture, gait-disorders-neurologic, hemiplegia, leprosy, malnutrition, neoplasms, nerve-compression-syndrome, nerve-damage, nerve-paralysis, numbness, pain, peripheral-neuropathy, peroneal-nerve-paralysis, peroneal-neuropathies, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Foot-drop, and have been seen in publications frequently: Aging, Dehiscence, Fermentation, Innervation, Localization, Locomotion, Menstruation, Muscle Atrophy, Muscle Contraction, Operant Conditioning, Ossification, Pathogenesis, Phagocytosis, Proprioception, Reflex, Schwann Cell Proliferation, Transport, Transposition, Vasoconstriction, Wound Healing
Quite a number of genes have been found to play important roles in Foot-drop, such as CERS2, CP, CST7, DBNL, FES, GDI1, GJB1, GNE, NLN, PES1, PMP22, PXMP2, RPL3, RPL5, SEPSECS, SERPINA5, SHANK2, TTN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.