Disease Info Card

Focal Nodular Hyperplasia

Information about Focal Nodular Hyperplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Focal Nodular Hyperplasia

Most recent studies have shown that Focal Nodular Hyperplasia shares some biological mechanisms with adenoma, benign-neoplasm, carcinoma, cholangiocarcinoma, focal-nodular-hyperplasia-of-the-liver, hemangioma, hemorrhage, hepatic-adenoma, hepatitis, hepatocellular-adenoma, hyperplasia, liver-carcinoma, liver-cirrhosis, liver-diseases, liver-neoplasms, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasm-metastasis, neoplasms, nodular-regenerative-hyperplasia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Focal Nodular Hyperplasia, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Growth, Cell Proliferation, Cell-cell Adhesion, Coagulation, Dedifferentiation, Enucleation, Excretion, Hemostasis, Liver Regeneration, Localization, Menopause, Methylation, Oncogenesis, Pathogenesis, Regeneration, Secretion, Transport

Quite a number of genes have been found to play important roles in Focal Nodular Hyperplasia, such as ACAT1, AFP, ALB, AR, CD34, F2R, FAM126A, FUT2, GLUL, GPC3, HNF1A, KRT19, KRT7, SLC25A5, SQLE, TP53, TRIM26. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.