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- Table of Contents
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Disease Info Card
Fatigability
Information about Fatigability: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fatigability
Most recent studies have shown that Fatigability shares some biological mechanisms with atrophy, autoimmune-diseases, autoimmune-reaction, blepharoptosis, depressive-disorder, dyspnea, headache, multiple-sclerosis, muscle-twitch, muscle-weakness, muscular-atrophy, myasthenia-gravis, myasthenias, myopathy, neoplasms, nervousness, neuromuscular-diseases, pain, sclerosis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fatigability, and have been seen in publications frequently: Aging, Coagulation, Electron Transport, Excretion, Flight, Glycolysis, Innervation, Localization, Menstruation, Muscle Atrophy, Muscle Contraction, Muscle Hypertrophy, Oxidative Phosphorylation, Pathogenesis, Reflex, Regeneration, Secretion, Skeletal Muscle Atrophy, Swimming, Transport
Quite a number of genes have been found to play important roles in Fatigability, such as ACHE, BLVRB, CAT, CRAT, CS, CSH1, CYCS, ERCC8, FUT2, GLYAT, HLA-E, HSPA9, IGHM, LIPG, MSLN, POMC, SLC4A1, SQLE, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.