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Disease Info Card
Familial Erythrocytosis
Information about Familial Erythrocytosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Erythrocytosis
Most recent studies have shown that Familial Erythrocytosis shares some biological mechanisms with anemia, erythrocytosis, familial-polycythemia-vera, hypoxia, leukemia, myeloid-leukemia-chronic, myeloproliferative-disease, neoplasms, paraganglioma, polycythemia, polycythemia-vera, primary-myelofibrosis, secondary-polycythemia, thrombocythemia-essential, thrombocytosis, tumor-angiogenesis, von-hippel-lindau-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Erythrocytosis, and have been seen in publications frequently: Angiogenesis, Blood Coagulation, Cell Growth, Cell Proliferation, Coagulation, Dna Replication, Excretion, Glycolysis, Hemostasis, Hypersensitivity, Methylation, Oxygen Homeostasis, Oxygen Transport, Pathogenesis, Response To Erythropoietin, Secretion, Translation, Transport
Quite a number of genes have been found to play important roles in Familial Erythrocytosis, such as ARHGEF7, ASCC1, CD177, CD40, EGLN1, EPAS1, EPO, EPOR, EPX, HIF1A, IL3, JAK2, NFKB1, PTPN6, STAT5A, TIMP1, VHL. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.