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Disease Info Card
Facial Paralysis
Information about Facial Paralysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Facial Paralysis
Most recent studies have shown that Facial Paralysis shares some biological mechanisms with acoustic-neuroma, bell-palsy, complete-hearing-loss, ear-inflammation, edema, facial-nerve-diseases, facial-nerve-injuries, facial-paresis, herpes-zoster-disease, lower-motor-neuron-facial-palsy, neoplasms, nerve-paralysis, neurilemmoma, ophthalmoplegia, otitis-media, paresis, parotid-neoplasms, peripheral-facial-palsy, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Facial Paralysis, and have been seen in publications frequently: Aging, Coagulation, Dehiscence, Enucleation, Hemostasis, Immune Response, Inflammatory Response, Innervation, Localization, Mastication, Muscle Atrophy, Muscle Contraction, Ossification, Pathogenesis, Reflex, Regeneration, Secretion, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Facial Paralysis, such as ABR, CAT, CD1B, CD1E, CD82, CDCA7L, CPA1, CRAT, CSF2, GLYAT, LAMC2, POMC, PRF1, RRM1, RRM2, TGM1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.