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Disease Info Card
Extrapulmonary Tuberculosis
Information about Extrapulmonary Tuberculosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Extrapulmonary Tuberculosis
Most recent studies have shown that Extrapulmonary Tuberculosis shares some biological mechanisms with abscess, acquired-immunodeficiency-syndrome, active-tuberculosis, aids-related-opportunistic-infections, granuloma, hiv-infections, immunologic-deficiency-syndromes, infective-disorder, lymph-node-tuberculosis, lymphadenitis, meningitis, pain, pleural-tuberculosis, tuberculosis, tuberculosis-gastrointestinal, tuberculosis-meningeal, tuberculosis-miliary, tuberculosis-osteoarticular, tuberculosis-pulmonary, tuberculosis-urogenital.
Among the many pathways, these few ones have gauged particular interests from scientists studying Extrapulmonary Tuberculosis, and have been seen in publications frequently: Adaptive Immune Response, Aging, Blood Circulation, Coagulation, Cytokine Production, Dna Amplification, Drug Resistance, Flight, Granuloma Formation, Humoral Immune Response, Hypersensitivity, Immune Response, Localization, Lymphocyte Proliferation, Macrophage Activation, Pathogenesis, Phagocytosis, Secretion, Segmentation, Virulence
Quite a number of genes have been found to play important roles in Extrapulmonary Tuberculosis, such as ADA, ALB, CD4, CSF2, HPD, IFNG, IL10, LAMC2, MUC5AC, NCAPG2, P2RX7, PTBP1, PTBP2, TNF, TST, VSX1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.