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Disease Info Card
Endotoxic Shock
Information about Endotoxic Shock: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Endotoxic Shock
Most recent studies have shown that Endotoxic Shock shares some biological mechanisms with acidosis, bacterial-infections, disseminated-intravascular-coagulation, edema, escherichia-coli-infections, hemorrhage, hypoglycemia, hypotension-adverse-event, infective-disorder, inflammation, inflammatory-response, ischemia, multiple-organ-failure, peripheral-circulatory-failure, salmonella-infections, septic-shock, septicemia, shock-hemorrhagic, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Endotoxic Shock, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Death, Chemotaxis, Coagulation, Complement Activation, Cytokine Production, Gluconeogenesis, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Phagocytosis, Platelet Aggregation, Secretion, Sensitization, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Endotoxic Shock, such as ALB, CD14, IFNG, IL10, IL1B, IL6, ISYNA1, LBP, MPO, NANOS2, NDUFA2, NFKB1, NOS2, POMC, SGSM3, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.