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Disease Info Card
Peripheral Circulatory Failure
Information about Peripheral Circulatory Failure: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Peripheral Circulatory Failure
Most recent studies have shown that Peripheral Circulatory Failure shares some biological mechanisms with cardiac-arrest, dengue-fever, dengue-hemorrhagic-fever, edema, heart-failure, hemorrhage, hemorrhagic-fevers-viral, hypotension-adverse-event, infective-disorder, ischemia, multiple-organ-failure, pain, septic-shock, staphylococcal-infections, streptococcal-infections, systemic-infection, toxic-shock-syndrome, toxic-shock-syndrome-streptococcal.
Among the many pathways, these few ones have gauged particular interests from scientists studying Peripheral Circulatory Failure, and have been seen in publications frequently: Anaphylaxis, Cell Activation, Cell Death, Cell Proliferation, Coagulation, Cytokine Production, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Menstruation, Pathogenesis, Reflex, Secretion, T Cell Activation, T Cell Proliferation, Transport, Vasoconstriction, Vasodilation, Virulence
Quite a number of genes have been found to play important roles in Peripheral Circulatory Failure, such as ALB, CFD, FUT2, GALNS, GAST, IFNG, IL10, IL1B, IL2, IL6, MPZ, NOS2, NR0B1, PAGR1, PMP22, SETBP1, SGSM3, SLC9A6, SQLE, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.