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Information about Deficiency Of Butyryl-coa Dehydrogenase: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Deficiency Of Butyryl-coa Dehydrogenase shares some biological mechanisms with acute-coronary-syndrome, acute-myocardial-infarction, aneurysm-dissecting, angina-pectoris, atherosclerosis, cerebrovascular-accident, coronary-aneurysm, coronary-artery-disease, dissection-of-coronary-artery, inborn-errors-of-metabolism, infarction, ischemia, long-chain-acyl-coa-dehydrogenase-deficiency, medium-chain-acyl-coenzyme-a-dehydrogenase-deficiency, multiple-acyl-coenzyme-a-dehydrogenase-deficiency, myocardial-infarction, pain, vertebral-artery-dissection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Deficiency Of Butyryl-coa Dehydrogenase, and have been seen in publications frequently: Aging, Cell Activation, Cell Death, Conjugation, Electron Transport, Electron Transport Chain, Excretion, Fatty Acid Beta-oxidation, Fatty Acid Oxidation, Inflammatory Response, Lipid Storage, Localization, Pathogenesis, Programmed Cell Death, Protein Folding, Translation, Transport, Tricarboxylic Acid Cycle, Urea Cycle, Vasodilation
Quite a number of genes have been found to play important roles in Deficiency Of Butyryl-coa Dehydrogenase, such as ACADL, ACADM, ACADS, ACADVL, ACCS, ACSS2, AKR1A1, CALD1, CDH15, CRP, DLD, ETFA, ITGB2, IVD, MTHFR, MUC1, OTC, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.