Disease Info Card

Closed Head Injuries

Information about Closed Head Injuries: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Closed Head Injuries

Most recent studies have shown that Closed Head Injuries shares some biological mechanisms with amnesia, brain-concussion, brain-damage-chronic, brain-edema, brain-injuries, cerebral-hemorrhage, cognition-disorders, comatose, contusions, craniocerebral-trauma, edema, fracture, hematoma, hematoma-subdural, hemorrhage, malnutrition, nonpenetrating-wounds, sequelae-aspects, skull-fractures, traumatic-brain-injury.

Among the many pathways, these few ones have gauged particular interests from scientists studying Closed Head Injuries, and have been seen in publications frequently: Cell Death, Coagulation, Cognition, Enucleation, Excretion, Inflammatory Response, Localization, Long-term Memory, Neuroprotection, Ossification, Pathogenesis, Reflex, Regeneration, Righting Reflex, Secretion, Social Behavior, Swimming, Translation, Transport, Visual Perception

Quite a number of genes have been found to play important roles in Closed Head Injuries, such as ALB, APOE, APP, AR, ASRGL1, AVP, CAT, CSF2, EPO, F11, F2, GCLC, GFAP, ICA, IL6, LAMC2, PTCRA, TNF, TP63, UGCG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.