This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Corneal Erosion
Information about Corneal Erosion: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Corneal Erosion
Most recent studies have shown that Corneal Erosion shares some biological mechanisms with cataract, cicatrix, conjunctival-diseases, conjunctivitis, corneal-diseases, corneal-ulcer, dystrophy, edema, eye-injuries, glaucoma, hereditary-corneal-dystrophy, inflammation, intraocular-pressure-disorder, keratitis, pain, recurrence-(disease-attribute), recurrent-erosion-of-cornea, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Corneal Erosion, and have been seen in publications frequently: Cell Adhesion, Cell Division, Cell Migration, Cell Proliferation, Delamination, Enucleation, Epithelial Cell Proliferation, Granuloma Formation, Inflammatory Response, Localization, Muscle Atrophy, Pathogenesis, Pigmentation, Reflex, Regeneration, Rna Splicing, Secretion, Tear Secretion, Virulence, Wound Healing
Quite a number of genes have been found to play important roles in Corneal Erosion, such as A4GALT, BBS9, DAPK3, DLK1, EGF, EGFR, FN1, IL5, IL6, IL7, MMP2, MMP9, MPO, OCA2, PLK3, PTK2B, TACSTD2, TGFBI. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.