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Disease Info Card
Corneal Dystrophy, Lattice Type 3
Information about Corneal Dystrophy, Lattice Type 3: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Corneal Dystrophy, Lattice Type 3
Most recent studies have shown that Corneal Dystrophy, Lattice Type 3 shares some biological mechanisms with amyloid-deposition, amyloidosis, amyloidosis-familial, blind-vision, corneal-diseases, corneal-edema, disorder-of-eye, dystrophy, eyelid-diseases, familial-amyloid-polyneuropathy-type-v, hereditary-corneal-dystrophy, localized-amyloidosis, plaque-amyloid, secondary-amyloidosis, trichiasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Corneal Dystrophy, Lattice Type 3, and have been seen in publications frequently: Enucleation, Localization, Pathogenesis, Spermatogenesis, Translation
Quite a number of genes have been found to play important roles in Corneal Dystrophy, Lattice Type 3, such as APCS, CLDN1, CLDN7, CLU, EGF, FGF7, GSN, HGF, KRT3, PSMD4, TACSTD2, TEAD1, TGFB1, TGFBI, TJP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Corneal Dystrophy, Lattice Type 3
This information is being compiled and will come in a future update
| Enucleation | Localization | Pathogenesis |
| Spermatogenesis | Translation |