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Disease Info Card
Contact Hypersensitivity
Information about Contact Hypersensitivity: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Contact Hypersensitivity
Most recent studies have shown that Contact Hypersensitivity shares some biological mechanisms with allergy, auricular-swelling, autoimmune-reaction, contact-dermatitis, decreased-immunologic-activity-[pe], delayed-hypersensitivity, dermatitis, dermatitis-allergic-contact, dermatitis-atopic, dermatologic-disorders, edema, erythema, inflammation, inflammatory-response, malignant-neoplasm-of-skin, malignant-neoplasms, neoplasms, skin-neoplasms, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Contact Hypersensitivity, and have been seen in publications frequently: Anaphylaxis, Cell Activation, Cell Adhesion, Cell Migration, Cell Proliferation, Chemotaxis, Cytokine Production, Dendritic Cell Migration, Dna Repair, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Pathogenesis, Secretion, Sensitization, T Cell Activation, T Cell Proliferation, Tolerance Induction
Quite a number of genes have been found to play important roles in Contact Hypersensitivity, such as APC, CD4, CD80, CD86, CD8A, CTLA4, DCX, HLA-DQA1, ICAM1, IFNG, IL10, IL1B, IL2, IL4, IL6, LYST, NOD2, POMC, PROC, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.