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- Table of Contents
Information about Congenital Stationary Night Blindness: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Congenital Stationary Night Blindness shares some biological mechanisms with achromatopsia, amaurosis, atrophy, blind-vision, congenital-night-blindness, disorder-of-eye, dystrophy, eye-diseases-hereditary, genetic-diseases-x-linked, leber-congenital-amaurosis, myopia, night-blindness, nystagmus, retinal-degeneration, retinal-diseases, retinitis-pigmentosa, retinoschisis, strabismus, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Stationary Night Blindness, and have been seen in publications frequently: Aging, Axon Guidance, Cell Adhesion, Cell Communication, Cell Maturation, Developmental Process, Immune Response, Localization, Mating, Membrane Depolarization, Pathogenesis, Phototransduction, Pigmentation, Regeneration, Secretion, Segmentation, Signal Transmission, Synaptic Transmission, Translation, Transport
Quite a number of genes have been found to play important roles in Congenital Stationary Night Blindness, such as CABP4, CACNA1F, ERG, GNAT1, GRK1, GRM6, KCNH2, NYX, PDE6B, RHO, RHOD, RPE65, RPGR, TNFSF14, TRPM1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.