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- Table of Contents
Information about Congenital Glucose-galactose Malabsorption: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Congenital Glucose-galactose Malabsorption shares some biological mechanisms with celiac-disease, chronic-diarrhea, congenital-abnormality, cystic-fibrosis, dehydration, diabetes-mellitus, diarrhea, diarrhea-watery, failure-to-thrive, fanconi-syndrome, gastroenteritis, glycosuria-renal, impaired-glucose-tolerance, infantile-diarrhea, lactose-intolerance, malabsorption-syndrome, malnutrition, nephrocalcinosis, severe-diarrhea.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Glucose-galactose Malabsorption, and have been seen in publications frequently: Amino Acid Transport, Excretion, Exocytosis, Fructose Transport, Galactose Transport, Glucose Homeostasis, Glucose Transport, Glycosylation, Hexose Transport, Insulin Secretion, Intestinal Absorption, Pathogenesis, Regulation Of Glucose Homeostasis, Secretion, Transepithelial Transport, Transport, Water Transport
Quite a number of genes have been found to play important roles in Congenital Glucose-galactose Malabsorption, such as AMY2A, C1QTNF1, EPHA3, GLS, ID1, INS, LCT, PSMD1, SLC2A1, SLC2A2, SLC2A4, SLC2A5, SLC5A1, SLC5A2, SLC5A4, SLC5A7. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.