Disease Info Card

Congenital Failure Of Fusion

Information about Congenital Failure Of Fusion: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Congenital Failure Of Fusion

Most recent studies have shown that Congenital Failure Of Fusion shares some biological mechanisms with cleft-lip, cleft-palate, cleft-palate-with-cleft-lip, congenital-abnormality, diastematomyelia, hydrocephalus, lipoma, meningocele, meningomyelocele, neoplasms, neurogenic-urinary-bladder, pain, scoliosis-unspecified, spina-bifida, spina-bifida-occulta, spinal-cord-diseases, spinal-cord-neoplasms, spinal-neoplasms, syringomyelia, tethered-spinal-cord-syndrome.

Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Failure Of Fusion, and have been seen in publications frequently: Brain Development, Cell Migration, Defecation, Dehiscence, Fertilization, Gastrulation, Innervation, Localization, Micturition, Muscle Atrophy, Neural Tube Closure, Ossification, Palate Development, Pathogenesis, Pigmentation, Reflex, Segmentation, Transposition, Tube Closure, Wound Healing

Quite a number of genes have been found to play important roles in Congenital Failure Of Fusion, such as C2, C6, CERS2, CIC, CSF2, HNRNPC, LAMC2, LPA, PES1, PPFIBP1, RANGAP1, RPL3, RPL4, RPL5, SEPSECS, SS18L1, ZACN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.