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Disease Info Card
Microscopic Polyangiitis
Information about Microscopic Polyangiitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Microscopic Polyangiitis
Most recent studies have shown that Microscopic Polyangiitis shares some biological mechanisms with anti-neutrophil-cytoplasmic-antibody-associated-vasculitis, arteritis, autoimmune-diseases, autoimmune-reaction, churg-strauss-syndrome, crescentic-glomerulonephritis, glomerulonephritis, hemorrhage, inflammation, kidney-diseases, kidney-failure, lung-diseases, lupus-erythematosus-systemic, necrotizing-vasculitis, polyarteritis-nodosa, purpura, vasculitis, wegener-granulomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Microscopic Polyangiitis, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Proliferation, Coagulation, Complement Activation, Endothelial Cell Activation, Fertilization, Fibroblast Proliferation, Glomerular Filtration, Glycosylation, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neutrophil Activation, Pathogenesis, Phagocytosis, Respiratory Burst, T Cell Activation
Quite a number of genes have been found to play important roles in Microscopic Polyangiitis, such as ACR, C3, CMAS, CP, CRP, CS, CSRP1, CTLA4, CTSG, CYCS, ELANE, ESR1, GYPA, IL2, IL5, MPO, PRTN3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.