Disease Info Card

Cerebrovascular Spasm

Information about Cerebrovascular Spasm: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Cerebrovascular Spasm

Most recent studies have shown that Cerebrovascular Spasm shares some biological mechanisms with aneurysm, aneurysm-ruptured, arterial-spasm, brain-ischemia, cerebral-infarction, cerebral-ischemia, hemorrhage, hemorrhage-aneurysmal-subarachnoid, hypertensive-disease, infarction, intracranial-aneurysm, ischemia, pathologic-vasoconstriction, rupture-spontaneous, spasm, stenosis, subarachnoid-hemorrhage, transient-ischemic-attack, vasospasm, vasospasm-intracranial.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cerebrovascular Spasm, and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Proliferation, Coagulation, Diuresis, Fibrinolysis, Inflammatory Response, Innervation, Localization, Membrane Depolarization, Muscle Contraction, Natriuresis, Neuroprotection, Pathogenesis, Platelet Aggregation, Reflex, Secretion, Smooth Muscle Contraction, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Cerebrovascular Spasm, such as ACSM3, AGT, ATP6V0A2, CAT, CHM, CSF2, EDN1, F2, GPHA2, ICA, LAMC2, NOS2, NOS3, PRRT2, SGCA, TTF2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.