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- Table of Contents
Information about Carotid Body Paraganglioma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Carotid Body Paraganglioma shares some biological mechanisms with carcinoma, cerebrovascular-accident, cranial-nerve-neoplasms, extra-adrenal-paraganglioma, glomus-jugulare-tumor, glomus-tumor, head-and-neck-neoplasms, hemorrhage, hypertensive-disease, lymphatic-metastasis, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasm-metastasis, neoplasm-recurrence-local, neoplasms, neoplasms-multiple-primary, neurilemmoma, paraganglioma, pheochromocytoma, thyroid-neoplasm.
Among the many pathways, these few ones have gauged particular interests from scientists studying Carotid Body Paraganglioma, and have been seen in publications frequently: Catecholamine Secretion, Cell Activation, Coagulation, Electron Transport, Electron Transport Chain, Excretion, Flight, Hemostasis, Hypersensitivity, Inflammatory Response, Innervation, Localization, Pathogenesis, Programmed Cell Death, Reflex, Response To Hypoxia, Secretion, Transport, Transposition, Vasodilation
Quite a number of genes have been found to play important roles in Carotid Body Paraganglioma, such as CALCA, ENO1, ENO2, GLYAT, ICA, OPN1SW, SDHAF2, SDHB, SDHC, SDHD, SDS, SDSL, SS18L1, SST, SYP, VEGFA, VHL. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.