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Disease Info Card
Carcinoma, Lewis Lung
Information about Carcinoma, Lewis Lung: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Carcinoma, Lewis Lung
Most recent studies have shown that Carcinoma, Lewis Lung shares some biological mechanisms with adenocarcinoma, b16-malignant-melanoma, carcinoma, cell-invasion, leukemia, lung-neoplasms, malignant-neoplasm-of-lung, malignant-neoplasms, malignant-paraganglionic-neoplasm, melanoma, melanoma-experimental, metastatic-malignant-neoplasm-to-the-lung, neoplasm-metastasis, neoplasms, neoplasms-experimental, pathologic-neovascularization, primary-neoplasm, sarcoma, tumor-angiogenesis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Carcinoma, Lewis Lung, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Killing, Cell Migration, Cell Motility, Cell Proliferation, Endothelial Cell Proliferation, Hypersensitivity, Immune Response, Induction Of Apoptosis, Localization, Macrophage Activation, Platelet Aggregation, Secretion, Sensitization, Transport, Tube Formation
Quite a number of genes have been found to play important roles in Carcinoma, Lewis Lung, such as CD4, CD8A, COL18A1, CSF2, DCX, FGF2, HBA1, IFNG, IL10, IL2, IL4, IL6, KDR, MMP2, MMP9, PECAM1, PLAU, PLG, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.