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Disease Info Card
Camurati-engelmann Syndrome
Information about Camurati-engelmann Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Camurati-engelmann Syndrome
Most recent studies have shown that Camurati-engelmann Syndrome shares some biological mechanisms with albers-schonberg-disease, anemia, bone-diseases, bone-diseases-developmental, complete-hearing-loss, dysplasia, exostoses, fracture, hyperostosis, muscle-weakness, muscular-dystrophy, neoplasms, osteochondrodysplasias, osteosclerosis, pain, ribbing-disease, sclerosis, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Camurati-engelmann Syndrome, and have been seen in publications frequently: Bone Development, Bone Remodeling, Bone Resorption, Cell Growth, Cellular Localization, Excretion, Glycosylation, Intramembranous Ossification, Ion Homeostasis, Localization, Locomotion, Ossification, Osteoclast Differentiation, Pathogenesis, Protein Glycosylation, Secretion, Spermatogenesis, Sulfation, Wound Healing
Quite a number of genes have been found to play important roles in Camurati-engelmann Syndrome, such as BGLAP, CALCA, CEACAM1, IFT122, LRP5, MTSS1, SLC26A2, SOST, TBXAS1, TGFB1, TGFBR1, TGFBR2, TNFRSF11A, TNFRSF11B, TNFSF11. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.