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Disease Info Card
Brucella Abortus Infection
Information about Brucella Abortus Infection: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Brucella Abortus Infection
Most recent studies have shown that Brucella Abortus Infection shares some biological mechanisms with bacterial-infections, brucella-melitensis-infection, brucellosis, cattle-diseases, communicable-diseases, decreased-immunologic-activity-[pe], delayed-hypersensitivity, infection-due-to-brucella-suis, infective-disorder, leptospirosis, neoplasms, pregnancy-complications-infectious, salmonella-infections, sheep-diseases, swine-diseases, tuberculosis, tuberculosis-bovine, yersinia-infections, zoonoses.
Among the many pathways, these few ones have gauged particular interests from scientists studying Brucella Abortus Infection, and have been seen in publications frequently: Cell Activation, Cell Cycle, Excretion, Hatching, Humoral Immune Response, Hypersensitivity, Immune Response, Lactation, Localization, Lymphocyte Proliferation, Macrophage Activation, Mating, Methylation, Parturition, Pathogenesis, Phagocytosis, Secretion, Sensitization, Transport, Virulence
Quite a number of genes have been found to play important roles in Brucella Abortus Infection, such as ALB, CD2, CD4, CD8A, EPHB1, HSPD1, IFNG, IL10, IL2, IL4, IL6, ITGA2, KCNH8, RPS19, SOD1, TLR2, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.