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- Table of Contents
Information about Branchial Clefts-congenital Disorder: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Branchial Clefts-congenital Disorder shares some biological mechanisms with abscess, branchioma, carcinoma, congenital-abnormality, cutaneous-fistula, dermoid-cyst, ectopic-tissue, edema, fistula-of-branchial-cleft, head-and-neck-neoplasms, lymphangioma, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, neoplasm-metastasis, neoplasms, parotid-neoplasms, pathologic-fistula, sinus-of-branchial-cleft-(disorder), thyroglossal-cyst.
Among the many pathways, these few ones have gauged particular interests from scientists studying Branchial Clefts-congenital Disorder, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Migration, Ear Development, Ear Morphogenesis, Glucose Transport, Inner Ear Development, Innervation, Keratinization, Kidney Development, Localization, Middle Ear Morphogenesis, Neural Crest Cell Migration, Pathogenesis, Pigmentation, Reflex, Transport
Quite a number of genes have been found to play important roles in Branchial Clefts-congenital Disorder, such as CDCA8, CDKN2A, CEACAM5, CKAP4, CRAT, EGF, EYA1, GLYAT, GSC, NKX2-1, RPE65, SERPINB3, SLC2A1, SS18L1, TG, THBS1, TTF1, UVRAG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.