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Information about Benign Mixed Epithelial And Stromal Tumor Of Kidney: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Benign Mixed Epithelial And Stromal Tumor Of Kidney shares some biological mechanisms with auricular-swelling, carcinoma, congenital-mesoblastic-nephroma, contact-dermatitis, cystic-nephroma, edema, epilepsy, infertility, kidney-neoplasm, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasms, neoplasms-complex-and-mixed, neoplasms-glandular-and-epithelial, nephroblastoma, pain, renal-cell-carcinoma, russell-silver-syndrome, stromal-neoplasm.
Among the many pathways, these few ones have gauged particular interests from scientists studying Benign Mixed Epithelial And Stromal Tumor Of Kidney, and have been seen in publications frequently: Cell Adhesion, Cell Proliferation, Demethylation, Dna Methylation, Embryo Development, Fertilization, Histone Modification, Hypersensitivity, Localization, Long-term Memory, Lymphocyte Proliferation, Maternal Behavior, Metaphase, Methylation, Oocyte Development, Oocyte Growth, Oogenesis, Pathogenesis, Sensitization, Spermatogenesis
Quite a number of genes have been found to play important roles in Benign Mixed Epithelial And Stromal Tumor Of Kidney, such as CDKN1C, COPG2, DES, DLK1, ESR1, GRB10, IGF2, IGF2R, MEST, NDN, PEG10, PEG3, PGR, PLAGL1, SNRPN, WDR20. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.