Disease Info Card

Amyloid Nephropathy

Information about Amyloid Nephropathy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Amyloid Nephropathy

Most recent studies have shown that Amyloid Nephropathy shares some biological mechanisms with amyloid-deposition, amyloidosis, arthritis, brucellosis, familial-mediterranean-fever, glomerulonephritis, hypertensive-disease, inflammation, kidney-diseases, kidney-failure, kidney-failure-chronic, multiple-myeloma, nephritis, nephrotic-syndrome, plaque-amyloid, primary-systemic-amyloidosis, proteinuria-of-undiagnosed-cause, rheumatoid-arthritis, secondary-amyloidosis, tuberculosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Amyloid Nephropathy, and have been seen in publications frequently: Acute-phase Response, Amyloid Fibril Formation, Cell Activation, Cell Adhesion, Cell Death, Cell Proliferation, Chemotaxis, Coagulation, Diuresis, Excretion, Glomerular Filtration, Hemostasis, Immune Response, Inflammatory Response, Localization, Pathogenesis, Protein Folding, Senescence, Spermatogenesis, Transport

Quite a number of genes have been found to play important roles in Amyloid Nephropathy, such as ALB, APCS, APOA1, APOA2, C3, CRP, CSRP1, HLA-DQA1, IL6, LYZ, MEFV, NLRP3, NOD2, SAA1, TEAD1, TNF, TNFRSF1A, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.