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Disease Info Card
Amebic Encephalitis
Information about Amebic Encephalitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Amebic Encephalitis
Most recent studies have shown that Amebic Encephalitis shares some biological mechanisms with acanthamoeba-infection, acanthamoeba-keratitis, acquired-immunodeficiency-syndrome, amebiasis, cns-disorder, encephalitis, granuloma, granulomatous-amebic-encephalitis, immunologic-deficiency-syndromes, infective-disorder, inflammation, keratitis, meningoencephalitis, meningoencephalitis-amoebic, meningoencephalitis-caused-by-naegleria-fowleri, nervousness, opportunistic-infections, pneumonia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Amebic Encephalitis, and have been seen in publications frequently: Aging, Autolysis, Cell Activation, Cell Cycle, Cell Death, Chemotaxis, Coenzyme Transport, Commensalism, Conjugation, Cytolysis, Humoral Immune Response, Immune Response, Inflammatory Response, Localization, Pathogenesis, Phagocytosis, Programmed Cell Death, Swimming, Transport, Virulence
Quite a number of genes have been found to play important roles in Amebic Encephalitis, such as ACAT1, ADK, ALB, CCNG1, CD6, CDK6, CNR1, CSF2, FN1, GADD45A, HM13, LAMC2, MCAT, MYCBP2, PAM, RB1, RBL2, SLC25A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.