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Disease Info Card
Alopecia Universalis
Information about Alopecia Universalis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Alopecia Universalis
Most recent studies have shown that Alopecia Universalis shares some biological mechanisms with acute-promyelocytic-leukemia, alopecia-areata, alopecia-totalis, atrichia, autoimmune-diseases, autoimmune-reaction, autoimmunity, congenital-abnormality, dermatitis, diabetes-mellitus, dystrophy, hashimoto-disease, hypothyroidism, immunologic-deficiency-syndromes, ophiasis, polyendocrinopathies-autoimmune, psoriasis, thyroiditis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Alopecia Universalis, and have been seen in publications frequently: Aging, Anagen, Cell Activation, Cell Cycle, Cytokine Production, Epithelium Development, Hypersensitivity, Immune Response, Interleukin-4 Production, Lymphocyte Proliferation, Menopause, Pathogenesis, Pigmentation, Proteolysis, Secretion, Sensitization, T Cell Activation, Telogen
Quite a number of genes have been found to play important roles in Alopecia Universalis, such as AIRE, CD4, CD8A, CTLA4, FOXP3, HLA-DQA1, HLA-DRB4, HR, IFNG, IL10, IL1RN, IL2, IL4, IL6, NOD2, POMC, TEAD1, TNF, VDR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.