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Disease Info Card
Acute Chest Syndrome
Information about Acute Chest Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acute Chest Syndrome
Most recent studies have shown that Acute Chest Syndrome shares some biological mechanisms with anemia, anemia-sickle-cell, asthma, cerebrovascular-accident, chest-pain, embolism, fat-embolism-as-early-complication-of-trauma-(disorder), hemoglobin-sc-disease, hemoglobinopathies, hypertensive-disease, infarction, infective-disorder, lung-diseases, pain, pneumonia, priapism, pulmonary-hypertension, thalassemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acute Chest Syndrome, and have been seen in publications frequently: Cell Adhesion, Chemotaxis, Coagulation, Glomerular Filtration, Hemostasis, Inflammatory Response, Lipid Transport, Membrane Organization, Neutrophil Activation, Oxygen Transport, Pathogenesis, Plasma Membrane Organization, Platelet Activation, Platelet Aggregation, Response To Bronchodilator, Response To Hydroxyurea, S Phase, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Acute Chest Syndrome, such as ACCS, ACSS2, ATP6V0A2, CRP, EDN1, ERMAP, GPHA2, HBB, HBG2, NOS2, NOS3, PLA2G10, PLA2G15, PLA2G2A, SCD, SGCA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.