Disease Info Card

Acquired Hyperostosis Syndrome

Information about Acquired Hyperostosis Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Acquired Hyperostosis Syndrome

Most recent studies have shown that Acquired Hyperostosis Syndrome shares some biological mechanisms with acne, ankylosing-spondylitis, arthritis, arthritis-psoriatic, chronic-multifocal-osteomyelitis, dermatologic-disorders, edema, hyperostosis, hyperostosis-sternocostoclavicular, inflammation, neoplasms, osteitis, osteomyelitis, pain, psoriasis, pustulosis-of-palms-and-soles, sclerosis, spondylarthropathies, spondylitis, synovitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Acquired Hyperostosis Syndrome, and have been seen in publications frequently: Bone Remodeling, Bone Resorption, Cell Proliferation, Chemotaxis, Cytokine Production, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neutrophil Chemotaxis, Ossification, Pathogenesis, Reflex, Respiratory Burst, Response To Antibiotic, Response To Tumor Necrosis Factor, T Cell Cytokine Production, Virulence

Quite a number of genes have been found to play important roles in Acquired Hyperostosis Syndrome, such as AHSG, CALCA, CD4, CD8A, CRP, CSRP1, ESR1, FGFR2, FUT2, IL1RN, INS, LPIN2, MRAP, NOD2, PSTPIP2, SQLE, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.