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Disease Info Card
Vasculitis, Leukocytoclastic, Cutaneous
Information about Vasculitis, Leukocytoclastic, Cutaneous: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Vasculitis, Leukocytoclastic, Cutaneous
Most recent studies have shown that Vasculitis, Leukocytoclastic, Cutaneous shares some biological mechanisms with arthritis, autoimmune-reaction, cryoglobulinemia, cutaneous-vasculitis, dermatologic-disorders, edema, erythema, exanthema, glomerulonephritis, hemorrhage, henoch-schoenlein-purpura, inflammation, lupus-erythematosus-systemic, polyarteritis-nodosa, purpura, ulcer, urticaria, vasculitis, wegener-granulomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Vasculitis, Leukocytoclastic, Cutaneous, and have been seen in publications frequently: Anaphylaxis, Cell Activation, Cell Adhesion, Chemotaxis, Coagulation, Complement Activation, Exocytosis, Fibrinolysis, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Mast Cell Degranulation, Pathogenesis, Pigmentation, Platelet Activation, Platelet Aggregation, Sensitization, Type Iii Hypersensitivity
Quite a number of genes have been found to play important roles in Vasculitis, Leukocytoclastic, Cutaneous, such as ACR, C3, C4A, CRP, CSF2, CSF3, ESR1, HLA-DQA1, ICAM1, IL6, ITGB2, MPO, PRTN3, SELE, SSB, TNF, TRIM21. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.