Disease Info Card

Uterine Corpus Intravenous Leiomyomatosis

Information about Uterine Corpus Intravenous Leiomyomatosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Uterine Corpus Intravenous Leiomyomatosis

Most recent studies have shown that Uterine Corpus Intravenous Leiomyomatosis shares some biological mechanisms with benign-neoplasm, endometrial-stromal-sarcoma, fibroid-tumor, heart-neoplasm, leiomyomatosis, leiomyosarcoma, malignant-paraganglionic-neoplasm, myoma, myomatous-neoplasm, neoplasm-invasiveness, neoplasm-metastasis, neoplasm-recurrence-local, neoplasms, sarcoma, smooth-muscle-tumor, syncope, uterine-fibroids, uterine-neoplasms, vascular-diseases, vascular-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Uterine Corpus Intravenous Leiomyomatosis, and have been seen in publications frequently: Angiogenesis, Menopause, Menstruation, Mitosis, Pathogenesis

Quite a number of genes have been found to play important roles in Uterine Corpus Intravenous Leiomyomatosis, such as AMY2A, CD34, CD99, CTLA4, DES, EPB42, ESR1, FGF2, HMGA2, IVL, LCP1, MME, PCNA, PGR, VEGFA, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Uterine Corpus Intravenous Leiomyomatosis Related Genes

click to see detail information for each gene

AMY2A CD34 CD99
CTLA4 DES EPB42
ESR1 FGF2 HMGA2
IVL LCP1 MME
PCNA PGR VEGFA
VIM

Pathways Related to Uterine Corpus Intravenous Leiomyomatosis

This information is being compiled and will come in a future update

Angiogenesis Menopause Menstruation
Mitosis Pathogenesis