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- Table of Contents
Information about True Hermaphroditism (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that True Hermaphroditism (disorder) shares some biological mechanisms with ambiguous-genitalia, chimera-disorder, congenital-abnormality, congenital-adrenal-hyperplasia, cryptorchidism, disorders-of-sex-development, embryonic-mosaic, feminization, gonadal-dysgenesis, gonadal-dysgenesis-mixed, gynecomastia, hermaphroditism, hypospadias, neoplasms, ovotesticular-disorders-of-sex-development, pseudohermaphroditism-male, sex-chromosome-aberrations, testicular-feminization, xx-male.
Among the many pathways, these few ones have gauged particular interests from scientists studying True Hermaphroditism (disorder), and have been seen in publications frequently: Cell Differentiation, Cell Division, Cell Proliferation, Fertilization, Gonad Development, Hatching, Localization, Mating, Meiosis, Oogenesis, Oviposition, Ovulation, Pathogenesis, Secretion, Sertoli Cell Differentiation, Sex Determination, Sex Differentiation, Spermatogenesis, Testosterone Secretion, Vitellogenesis
Quite a number of genes have been found to play important roles in True Hermaphroditism (disorder), such as AMH, AR, BRD2, CD38, CYP19A1, DMRT1, KDM5D, NOD2, NR5A1, PLOD1, SNAI2, SOX9, SRY, TH, WNT4, ZFY. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.