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Disease Info Card
Sensory Ataxia
Information about Sensory Ataxia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sensory Ataxia
Most recent studies have shown that Sensory Ataxia shares some biological mechanisms with absence-of-sensation, ataxia, atrophy, cerebellar-ataxia, demyelination, malignant-neoplasms, muscle-weakness, nervous-system-disorder, ophthalmoplegia, pain, paresthesia, peripheral-neuropathy, polyneuropathy, polyradiculoneuropathy, sensation-disorders, sensory-neuropathy, spinal-cord-diseases, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sensory Ataxia, and have been seen in publications frequently: Aging, Cell Activation, Cytoplasmic Translation, Diuresis, Fatty Acid Transport, Hyperphosphorylation, Intracellular Transport, Lipid Storage, Mitochondrial Translation, Muscle Atrophy, Myelination, Nuclear Export, Oxidative Phosphorylation, Pathogenesis, Proprioception, Reflex, Regeneration, Sensitization, Translation, Transport
Quite a number of genes have been found to play important roles in Sensory Ataxia, such as ABCD1, ACAT1, CHMP1B, CP, CSF2, DST, FXN, GAA, LAMC2, MAG, MPZ, NTF3, POLG, SLC25A5, UCHL1, UCHL3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.