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Disease Info Card
Renal Osteodystrophy
Information about Renal Osteodystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Renal Osteodystrophy
Most recent studies have shown that Renal Osteodystrophy shares some biological mechanisms with bone-diseases, calcinosis, chronic-kidney-disease, fracture, hypercalcemia, hyperparathyroidism-secondary, hyperphosphatemia, kidney-diseases, kidney-failure, kidney-failure-chronic, metabolic-bone-disorder, osteitis-fibrosa-cystica, osteitis-fibrosa-disseminata, osteomalacia, osteoporosis, renal-insufficiency, rickets, uremia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Renal Osteodystrophy, and have been seen in publications frequently: Aging, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Growth, Cell Proliferation, Chondrocyte Proliferation, Diuresis, Excretion, Glomerular Filtration, Hormone Secretion, Intestinal Absorption, Localization, Menopause, Ossification, Osteoblast Differentiation, Parathyroid Hormone Secretion, Pathogenesis, Secretion, Transport
Quite a number of genes have been found to play important roles in Renal Osteodystrophy, such as ALB, ALPP, ASRGL1, ATRNL1, BEST1, BGLAP, CALCA, CCL27, DMD, FGF23, HTT, KNTC1, PDLIM3, PTH, PTRH1, SLPI, SS18L1, TNFRSF11B. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.