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Information about Refractory Anemia With Excess Blasts In Transformation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Refractory Anemia With Excess Blasts In Transformation shares some biological mechanisms with acute-leukemia, anemia, cytogenetic-abnormality, dysmyelopoietic-syndromes, dysplasia, graft-vs-host-disease, leukemia, leukemia-myelocytic-acute, leukemia-myelomonocytic-chronic, malignant-neoplasms, monosomy, myeloid-leukemia, myelomonocytic-leukemia, neoplasms, pancytopenia, refractory-anaemia-with-excess-blasts, refractory-anemia-with-ringed-sideroblasts, refractory-anemias, trisomy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Refractory Anemia With Excess Blasts In Transformation, and have been seen in publications frequently: Angiogenesis, Cell Activation, Cell Adhesion, Cell Cycle, Cell Death, Cell Growth, Cell Proliferation, Chemotaxis, Dna Repair, Gene Silencing, Hemopoiesis, Localization, Metaphase, Methylation, Neutrophil Differentiation, Oncogenesis, Pathogenesis, Phagocytosis, Programmed Cell Death, Response To Erythropoietin
Quite a number of genes have been found to play important roles in Refractory Anemia With Excess Blasts In Transformation, such as ASXL1, CD34, CDKN2B, CSF2, CSF3, CTLA4, EPO, FANCB, HLA-DQA1, IL3, NOD2, PAFAH1B1, SF3B1, TNF, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.