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Disease Info Card
Presbycusis
Information about Presbycusis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Presbycusis
Most recent studies have shown that Presbycusis shares some biological mechanisms with abnormal-degeneration, atrophy, complete-hearing-loss, conductive-hearing-loss, diabetes-mellitus, ear-diseases, ear-inflammation, hearing-loss-bilateral, hearing-loss-high-frequency, hearing-problem, hypertensive-disease, impairment-(finding), labyrinthine-disorder, malnutrition, meniere-disease, nerve-degeneration, noise-induced-hearing-loss, otitis-media, otosclerosis, sensorineural-hearing-loss-(disorder).
Among the many pathways, these few ones have gauged particular interests from scientists studying Presbycusis, and have been seen in publications frequently: Aging, Cell Death, Cell Proliferation, Coagulation, Cognition, Habituation, Innervation, Ion Homeostasis, Ion Transport, Localization, Oxidative Phosphorylation, Pathogenesis, Prepulse Inhibition, Reflex, Regeneration, Response To Oxidative Stress, Senescence, Sensory Processing, Translation, Transport
Quite a number of genes have been found to play important roles in Presbycusis, such as ABR, CAP1, CASP3, CAST, CD3EAP, CDH23, COCH, DBI, DIRAS3, EPO, F11, GJB2, HES1, HMGCL, LIPC, PTCRA, SGPL1, SOD1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.