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Disease Info Card
Ocular Toxoplasmosis
Information about Ocular Toxoplasmosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ocular Toxoplasmosis
Most recent studies have shown that Ocular Toxoplasmosis shares some biological mechanisms with acquired-immunodeficiency-syndrome, anterior-uveitis, aqueous-humor-disorders, blind-vision, chorioretinitis, choroiditis, cicatrix, cytomegalovirus-infections, disorder-of-eye, infective-disorder, inflammation, pregnancy-complications-infectious, retinal-detachment, retinal-diseases, toxoplasmosis, toxoplasmosis-animal, toxoplasmosis-congenital, uveitis, uveitis-posterior.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ocular Toxoplasmosis, and have been seen in publications frequently: Acute Inflammatory Response, Aging, Cell Adhesion, Cell Cycle Phase, Coagulation, Cytokine Secretion, Dna Amplification, Enucleation, Fertilization, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Pigmentation, Reverse Transcription, Secretion, Sensitization, Virulence
Quite a number of genes have been found to play important roles in Ocular Toxoplasmosis, such as BBS9, CAT, CD4, CD8A, CRAT, GLYAT, IFNG, IL10, IL17A, IL2, IL4, IL6, MS4A1, RHCE, RHD, SPANXB1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.