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- Table of Contents
Information about Neutropenia Chronic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Neutropenia Chronic shares some biological mechanisms with agranulocytosis, anemia, arthritis, autoimmune-diseases, autoimmune-reaction, bacterial-infections, cyclic-neutropenia, dysmyelopoietic-syndromes, glycogen-storage-disease, infective-disorder, leukemia, leukemia-myelocytic-acute, malignant-paraganglionic-neoplasm, myeloid-leukemia, rheumatoid-arthritis, severe-congenital-neutropenia, storage-disease, t-cell-large-granular-lymphocyte-leukemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Neutropenia Chronic, and have been seen in publications frequently: Acute Inflammatory Response, Cell Cycle, Cell Maturation, Cell Proliferation, Chemotaxis, Glucose Homeostasis, Glucose-6-phosphate Transport, Immune Response, Inflammatory Response, Neutrophil Chemotaxis, Neutrophil Differentiation, Pathogenesis, Phagocytosis, Respiratory Burst, Ribosome Assembly, Ribosome Biogenesis, Rna Splicing, Sensitization, Superoxide Anion Generation, Transport
Quite a number of genes have been found to play important roles in Neutropenia Chronic, such as ASXL1, CD40LG, CSF2, CSF3, CSF3R, ELANE, EPO, G6PC, HSPA9, IL2, IL3, PAFAH1B1, SF3B1, SLC37A4, TNF, USB1, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.