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- Table of Contents
Information about Multiple Sulfatase Deficiency Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Multiple Sulfatase Deficiency Disease shares some biological mechanisms with deficiency-of-arylsulfatase, deficiency-of-sulfatase, gangliosidoses, gaucher-disease, hydrocephalus, ichthyoses, ichthyosis-x-linked, inborn-errors-of-metabolism, leukodystrophy, leukodystrophy-metachromatic, lysosomal-storage-diseases, mucolipidoses, mucopolysaccharidoses, mucopolysaccharidosis-iv, mucopolysaccharidosis-vi, nerve-degeneration, neurodegenerative-disorders, sphingolipidoses, storage-disease.
Among the many pathways, these few ones have gauged particular interests from scientists studying Multiple Sulfatase Deficiency Disease, and have been seen in publications frequently: Autophagy, Bone Development, Cell Death, Cell Development, Cellular Process, Endocytosis, Excretion, Exocytosis, Intracellular Transport, Lipid Storage, Localization, Lymphocyte Differentiation, Macroautophagy, Pathogenesis, Regeneration, Reverse Transcription, Secretion, Secretory Pathway, Translation, Transport
Quite a number of genes have been found to play important roles in Multiple Sulfatase Deficiency Disease, such as ABCB6, ARSA, ARSB, ARSH, BBS9, CTLA4, DEGS1, GALNS, HLA-DQA1, LMAN1, NOD2, SGSH, SLURP1, STS, SUMF1, SUMF2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.