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- Table of Contents
Information about Milroy Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Milroy Disease shares some biological mechanisms with aplasia-nos, congenital-abnormality, edema, elephantiasis, fibrosis, hyperplasia, hypoplasia, lymphangiectasis, lymphangiosarcoma, lymphatic-diseases, lymphedema, lymphedema-praecox, malignant-neoplasms, neoplasm-metastasis, neoplasms, pleural-effusion-disorder, secondary-lymphedema, venous-insufficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Milroy Disease, and have been seen in publications frequently: Acute Inflammatory Response, Aging, Angiogenesis, Cell Adhesion, Diuresis, Immune Response, Inflammatory Response, Keratinization, Localization, Lymph Circulation, Lymphangiogenesis, Menarche, Oncogenesis, Pathogenesis, Regeneration, Regulation Of Lymphangiogenesis, Transport, Transposition, Tube Formation, Vasculogenesis
Quite a number of genes have been found to play important roles in Milroy Disease, such as ALB, ANGPT1, FLT4, FMN1, FOXC1, FOXC2, GATA2, LYVE1, MTSS1, NRP2, PDPN, PGF, RELN, SOX18, VEGFA, VEGFC. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.