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Disease Info Card
Lysinuric Protein Intolerance
Information about Lysinuric Protein Intolerance: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lysinuric Protein Intolerance
Most recent studies have shown that Lysinuric Protein Intolerance shares some biological mechanisms with aminoaciduria, anemia, comatose, cystinuria, diarrhea, failure-to-thrive, hemorrhage, hyperammonemia, inborn-errors-of-metabolism, kidney-diseases, lung-diseases, metabolic-diseases, orotic-aciduria, osteoporosis, pulmonary-alveolar-proteinosis, renal-aminoacidurias, vomiting.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lysinuric Protein Intolerance, and have been seen in publications frequently: Amino Acid Transport, Arginine Transport, Cell Proliferation, Excretion, Granuloma Formation, Hormone Secretion, Immune Response, Inflammatory Response, Intestinal Absorption, Leucine Transport, Localization, Macrophage Activation, Pathogenesis, Phagocytosis, Secretion, Translation, Transport, Urea Cycle, Vasodilation, Wound Healing
Quite a number of genes have been found to play important roles in Lysinuric Protein Intolerance, such as ASL, ASS1, CTLA4, HLA-DQA1, IGF1, LAT, MTSS1, MYH7, NOD2, PYCARD, SLC3A2, SLC7A1, SLC7A11, SLC7A2, SLC7A5, SLC7A6, SLC7A7, SLC7A9, STS, TSHZ1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.