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Disease Info Card
Pulmonary Alveolar Proteinosis
Information about Pulmonary Alveolar Proteinosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pulmonary Alveolar Proteinosis
Most recent studies have shown that Pulmonary Alveolar Proteinosis shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, coughing, dyspnea, fibrosis, infective-disorder, inflammation, leukemia, lung-diseases, lung-diseases-interstitial, lung-neoplasms, malignant-neoplasms, pneumonia, pneumonia-interstitial, pulmonary-fibrosis, respiratory-distress, respiratory-failure, sarcoidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pulmonary Alveolar Proteinosis, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Cell Adhesion, Cell Proliferation, Chemotaxis, Cytokine Production, Glycosylation, Hypersensitivity, Immune Response, Inflammatory Response, Lipid Homeostasis, Localization, Macrophage Differentiation, Pathogenesis, Phagocytosis, Regeneration, Secretion, Surfactant Homeostasis, Translation, Transport
Quite a number of genes have been found to play important roles in Pulmonary Alveolar Proteinosis, such as ASAP1, ASAP2, CSF2, IL3, IL5, MRPS30, MUC1, PAPOLA, PDAP1, REG3A, SFTPB, SFTPC, SFTPD, TFF2, TUSC2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.