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Disease Info Card
Ichthyosis Vulgaris
Information about Ichthyosis Vulgaris: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ichthyosis Vulgaris
Most recent studies have shown that Ichthyosis Vulgaris shares some biological mechanisms with asthma, bullous-congenital-ichthyosiform-erythroderma-(disorder), congenital-nonbullous-ichthyosiform-erythroderma, dermatitis, dermatitis-atopic, dermatologic-disorders, eczema, exfoliative-dermatitis, hyperkeratosis, ichthyoses, ichthyosiform-erythroderma-congenital, ichthyosis-x-linked, keratosis, keratosis-follicularis, pruritus, psoriasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ichthyosis Vulgaris, and have been seen in publications frequently: Cell Activation, Cell Cycle, Cell Proliferation, Complement Activation, Cornification, Diuresis, Excretion, Hypersensitivity, Intestinal Absorption, Keratinization, Keratinocyte Differentiation, Lipid Storage, Localization, Pathogenesis, Pigmentation, Proteolysis, Secretion, Sensitization, Translation, Transport
Quite a number of genes have been found to play important roles in Ichthyosis Vulgaris, such as CDSN, DSG1, ERMAP, FGFR1, FLG, HSPB1, IGHE, IL13, IVL, KLK7, KRT1, KRT10, NOD2, PCNA, STS, TGM1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.