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Disease Info Card
Hyperphagia
Information about Hyperphagia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperphagia
Most recent studies have shown that Hyperphagia shares some biological mechanisms with depressive-disorder, diabetes-mellitus, diabetes-mellitus-experimental, eating-disorders, hyperglycemia, hyperinsulinism, insulin-resistance, malignant-neoplasms, malnutrition, neoplasms, nervousness, obesity, prader-willi-syndrome, vomiting.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperphagia, and have been seen in publications frequently: Aging, Cognition, Eating Behavior, Energy Homeostasis, Excretion, Fatty Acid Oxidation, Feeding Behavior, Gastric Emptying, Glucose Homeostasis, Hypersensitivity, Insulin Secretion, Lactation, Locomotion, Pathogenesis, Regulation Of Appetite, Regulation Of Energy Homeostasis, Response To Food, Secretion, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Hyperphagia, such as AGRP, ALB, ARC, BAAT, BDNF, CCK, CRH, GCG, INS, LEP, LEPR, MC4R, NOL3, NPY, POMC, PRL, PYY, ST3GAL4. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.